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Activation of GLP-1R also promotes beta cell survival, inhibits gastric emptying, regulates food intake and reduces appetite (1, 5), making it a key pharmacological target for various metabolic disorders including type 2 diabetes (T2D) and obesity (6, 7)

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Once secreted, both endogenous GIP and GLP-1 undergo rapid degradation into the biologically inactive metabolites (50, 51)

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Microdosing or starting with a l ower dose may help to prevent gastrointestinal side effects